Nr507 Week 5: Disease Process Presentation Assignment Part 2
The purpose of the Nr507 Week 5: Disease Process Assignment Part 2 is for learners to:
- Develop professional presentation/communication skills.
- Link development of initial presenting signs and symptoms of disease to changes to tissue, organ and system functioning.
- Describe how the disease is diagnosed
- Describe pharmacological and non-pharmacological interventions for disease management.
Due Date: Sunday 11:59 PM MT at the end of Week 5
Course Outcomes
- Analyze pathophysiological mechanisms associated with select disease states.
- Relate research findings to the management of patients with complex pathophysiologic dysfunction.
Total Points Possible: 100 points
Requirements
Description of the Assignment
In Week 7, students will present an audio/video PowerPoint presentation using the Disease Process Presentation Guidelines and Rubric located under Course Resources. To ensure timely development of the Week 7 assignment and an opportunity to receive feedback from the course faculty, students will develop the Week 7 presentation in 2 parts. Parts 1 and 2 will be submitted as a PowerPoint presentation that contains thorough speaker notes. After students have received faculty feedback on Parts 1 and 2, students will develop the final presentation that is audio and video recorded and uploaded to the Week 7 Discussion Board.
Criteria for Content
- Link changes in the tissue, organ, and system functioning to the initial presenting signs and symptoms seen in primary careof the disease.
- Provide a brief description of how the disease is diagnosed.
- Provide a brief description of the pharmacological and non-pharmacological interventions used to treat and manage the disease.
- Summarizes the disease on final slide with concluding remarks; includes implication for nurse practitioner practice.
- Utilizes at least two current (within 5 years), peer-review scholarly sources to support presentation content.
- Reference slide and in-text citations depict references correctly cited according to APA.
Nr507 Week 5: Disease Process Answer
Diabetes mellitus type 1 (DM1) is a chronic health condition characterized by insufficient insulin production in the pancreas, primarily due to autoimmune destruction of beta cells (Katsarou et al., 2017). Insulin plays a crucial role in facilitating the transfer of glucose from the bloodstream into cells for energy production. Although DM1 is more commonly diagnosed in children and adolescents, it can also manifest in adults.Nr507 Week 5: Disease Process
The digestive system metabolizes carbohydrates into glucose, which is then transported through the bloodstream to cells for energy utilization. The pancreas secretes insulin, enabling the movement of glucose from the blood into body cells (Zaccardi, Webb, Yates, & Davies, 2016). Within the cells, glucose undergoes combustion with oxygen, generating energy. In individuals with type 1 diabetes, insufficient insulin production disrupts this process, leading to elevated blood glucose levels and decreased glucose availability for cellular energy production (Katsarou et al., 2017).Nr507 Week 5: Disease Process
To compensate for the energy deficit, the body attempts to supply cells with alternative fuel sources, such as stored fats. The heightened utilization of fats results in the production of ketones as by-products, which, in excessive amounts, can pose life-threatening complications (Zaccardi et al., 2016).Nr507 Week 5: Disease Process
Understanding the intricate metabolic disruptions in type 1 diabetes is essential for comprehensive patient care, allowing healthcare professionals to tailor interventions that address both glucose management and potential complications associated with altered metabolic pathways.
References: Katsarou, A., Gudbjörnsdottir, S., Rawshani, A., Dabelea, D., Bonifacio, E., Anderson, B. J., Jacobsen, L. M., Schatz, D. A., Lernmark, Å., & Type 1 Diabetes Mellitus Natural History Consortium. (2017). Type 1 diabetes mellitus. Nature Reviews Disease Primers, 3, 17016. https://doi.org/10.1038/nrdp.2017.16
